![]() Philadelphia: Lippincott Williams & Wilkins. In: Lippincott's Pathophysiology Series: Pulmonary Pathophysiology. “Gas transport to and from peripheral tissues”. In: Fishman's Pulmonary Diseases and Disorders, edited by MA, Grippi (editor-in-chief), JA, Elias, JA, Fishman, RM, Kotloff, AI, Pack, RM, Senior (editors). ![]() “Clinical presentations: gas exchange and transport”. I literally know nothing more on the subject. Treat a pulmonary embolus or anything perfusion limited, assuming Pressure difference across HEALTHY lung (in this instance) willĬause a higher oxygen saturation than typical in the remainingĬapillary beds not blocked by the embolus. Good would adding more O2 do? Again, increasing the partial Shows equilibration of gases by the end of the capillary, so what This is tricky, because perfusion limitation In the case of a pulmonary embolism, there is Blasting them with O2 will do little.ĭead space or perfusion limitation on the other hand does YouĪre not doing anything to ameliorate the initial problem: bad Such people cannot be treated with 100% O2 very successfully. Participating in gas exchange for the reasons I just mentioned.Īlso think of pulmonary fibrosis, where thickness is increased. Surface area is diminished because of less healthy lung tissue Little fibrosis in emph., it's not a thickness issue). Partial pressure of the gas of interest on both sides (in the blood The surface area available for exchange, and the difference in Membrane/solubility of the gas, the thickness of this interface, There is a limitation to diffusion because theĮndothelial alveolar interface is destroyed in emphysema. Shunts are diffusion limited byĭefinition.
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